RANKL/RANK/MMP-1 molecular triad contributes to the metastatic phenotype of breast and prostate cancer cells in vitro.

of breast cancer associated with certain metastatic sites exhibit a characteristic genetic expression, and the immunohistochemical expression of the human epidermal growth factor

We investigated expression levels of MMP-2, MMP-9, COX-2, RANK/RANKL and SOCS-1 in rat intestinal tissues from the negative control, MTX , and MTX- OLM 5 mg/kg groups

The compounds were evaluated for their in vitro cytotoxicity activities against cultured human cancer cells of PC-3 human prostate cancer, MCF-7 and MDA-MB-231 breast carcinoma,

To investigate the mechanisms underlying the growth and survival-promoting effects of DCD in MDA-MB- 361 breast cancer cells, we analyzed the global gene expression profiles of

We uncovered the anti-metastatic potential of fisetin in cervical cancer cells and elucidated its molecular mechanism, that is fisetin inhibits the activation of p38

Malignant prostate cells express CXCL16 and CXCR6 In order to investigate the roles of chemokines in prostate cancer, we screened prostate cancer cell lines and xenografts

Comparison of HIF-1 α , PKM2 and ISCU1/2 protein expression in metastatic breast cancer cells and metastatic melanoma cells in the presence and absence of methyl sulfone and

Increased expression of Rev3 in human breast epithelial cancer cell lines compared to non- neoplastic human breast epithelial cells MCF-12A, and in human breast tumors compared