A Novel Missense Mutation of GATA4 in a Chinese Family with Congenital Heart Disease.

Here, we report that a novel ER membrane protein, FT-INTERACTING PROTEIN 1 (FTIP1), interacts with FT in companion cells of the phloem (a specialized type of parenchyma cell in

We screened the TBX5, GATA4, and NKX2.5 genes for mutations, by direct sequencing, in 32 unrelated patients presenting classical (8) or atypical HOS (1), isolated congenital

HeLa cells permanently transfected with a plasmid containing the gene encoding the expression of the 15 kDa protein enriched in astrocytes (PEA-15) were used to

In contrast, NO concentrations in the supernatants of spleen cells from mice immunized with the control plasmid pcDNA3 cultured in the presence or absence of the recombinant

To evaluate the effects of ectopic expression of individual TRIMs on HBV replication in human hepatocytes, HepG2 cells were co-transfected with a plasmid containing 1.3 fold

When comparing the two classroom types with Pearson’s chi-squared test, in the success-oriented classrooms it was more common to maintain the same achievement goal profile over time

Our findings demonstrate that GATA4 and its downstream targets are regulated in the heart of the hibernating thirteen-lined ground squirrel over the torpor-arousal cycle

Human HeLa and HOS cells, together with AGM Vero cells, were transfected with HIV-1 NL4.3 and NL4.3delVpu proviral plasmids, or an MLV Gag-Pol– expression vector, as indicated, in

TC620 cells were transfected with luciferase reporter plasmid for the early promoter, JCV E -LUC in the presence or absence of increasing amounts of Rad51 expression plasmid (0, 0.5