Split-cre complementation indicates coincident activity of different genes in vivo.

Transgenic mice carrying the human kallikrein gene under the control of the mouse metallothionein metal-responsive promoter expressed the human protein in several or- gans

To assess the effectiveness of the model we compared the generated solutions with the solutions generated via two other different methods: (1) a portfolio split

We generated DLC2-deficient mice to investigate the tumor suppressor role of DLC2 in hepatocarcinogenesis and the function of DLC2 in vivo.. In this study, we found that,

Cis -complementation of essential MCMV genes in cells In order to test the basic principle, Flpe-NIH were nucleofected with MCMV BACs which were generated by deletion of the

To investigate the mechanism of lymphoma regression, we tested whether Cre activation could induce apoptosis in a p53 2 / 2 lymphoma cell line derived from the mouse model.. Cre-ER

Using the Mx1-Cre;mT/mG reporter mouse model, we found that the calvarial cells exhibited minimal background Mx1-Cre activity prior to Cre activation by IFN α treatment as compared

To compare the normalized data from dysplasia, normal lung tissue from transgenic mouse, tumor cells and non-transgenic of different mice, we used the Significance Analysis

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