Myoferlin depletion in breast cancer cells promotes mesenchymal to epithelial shape change and stalls invasion.

The compounds were evaluated for their in vitro cytotoxicity activities against cultured human cancer cells of PC-3 human prostate cancer, MCF-7 and MDA-MB-231 breast carcinoma,

Examining cell behaviors of MCF-10A, MCF- 7, and MDA-MB-231 cells with our established platform showed that (a) the highly metastatic MDA-MB-231 breast cancer cells had the

signaling can partially mediate the anti-proliferative effect that ER a showed in the aggressive breast cancer cells, a genome-wide expression analysis in aggressive MDA-MB-231

indeed promotes EMT in these ovarian cancer cells, we examined the expression of E-cadherin and N-cadherin, two well-established epithelial to mesenchymal makers, in

The ability of the conserved regions to direct gene expression was tested using three previously characterized human breast cancer cells, MDA-MB-231, SUM159, and MCF7, each with

We compared the proliferation of the human breast cancer cell line MDA-MB-231 and of the non-cancer human mammary epithelial cell line MCF-10A in different experimental

In RNAi- mediated ADAM9 silenced MDA-MB-231 cells, the expression of ADAM9 was lower from the third to the sixth day after silencing and inhibited tumor cell invasion in matrigel

In RNAi- mediated ADAM9 silenced MDA-MB-231 cells, the expression of ADAM9 was lower from the third to the sixth day after silencing and inhibited tumor cell invasion in matrigel