MiR-132 suppresses the migration and invasion of lung cancer cells via targeting the EMT regulator ZEB2.

Besides, we observed that propofol stimulated the expression of miR-486, and suppression of miR-486 reversed the effects of propofol on lung cancer cell viability, apoptosis,

nels (VGSC) in the progression of different tumors, such as prostate cancer, small cell lung cancer, breast cancer and others, linking VGSC to the invasion capacity of tumor cells (4

bone metastasis is the result of complicated interactions be- tween tumor cells and various stromal cells in bone, leading to the initial survival of cancer cells in the

Its knockdown by siRNA decreased the proliferation, migration, and invasion of gastric cancer cells, whereas its over- expression showed the opposite effects.. These results

)GF R were identified as the regulated target gene in previous studies ,. )t is possible that the discrepancies in the functions of miR‐ in different types of cancer

To test whether ouabain was able to inhibit migration in other lung cancer cells, we treated the human non-small cell lung cancer cell line H460 with ouabain (0–30 pM) and subjected

Stratified analysis implies the association of the polymorphisms in mir-196a2 and mir-149 with NSCLC, while the association of the four polymorphisms with digestive cancer

These results indicate that miR-200c overexpression regulates MUC4 and MUC16 mucins in pancreatic cancer cells by directly targeting the mRNA coding sequence of