Neuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membrane.

Targeted disruption of the neuronal nitric oxide synthase (nNOS) and endothelial nitric oxide synthase (eNOS) genes has led to knockout mice that lack these isoforms.. These

Expression of neuronal nitric oxide synthase-immu- noreactive (nNOS-IR) neurons in the CA1 and subiculum (Sub) of hippocampal formation of schizophrenia (SCZ), major depres- sion

Using an animal model of FXS, FMR1 KO mice, we compared the expression of neuronal NOS (nNOS) mRNA and protein levels from the hippocampi of wild-type (WT) and FMR1 KO mice at

Serial muscle sections from normal BL10 ( A ), mdx ( B ), utrophin knockout ( C ), u-dko ( D ) and Fiona strain of utrophin transgenic mdx mice ( E ) were stained for general

Our results showed that ginsenoside Rg1 activated IR and the AMPK pathway in skeletal muscle and liver of HFD mice as well as increased GLUT4 translocation to the plasma membrane

Neste mesmo capítulo são discutidos os tipos de performance do guitarrista de acordo com os tipos de interface e equipamentos que lhe são disponibilizados pela

Inhibition of allergic airway inflammation in mice lacking nitric oxide synthase 2. Role of exhaled nitric oxide

Purpose: We studied the distribution of neuronal nitric oxide synthase (nNOS) and the effects of nitric oxide (NO) modulating drugs on contractile function of the external