Intestinal Stem Cell Markers in the Intestinal Metaplasia of Stomach and Barrett's Esophagus.

The presence of CaCCs in native intestinal epithelium has been more difficult to demonstrate and there is a body of evidence suggesting that, although calcium-activated

(1996) found significantly higher numbers of globule leucocytes/mucosal mast cells in the intestinal mucosa in resistant than in susceptible lambs that were naturally infected

and eosin (HE) showed a reducion in goblet cells difusely in the colon segment, and mild to moderate lat and papillary transiional hyperplasia was observed in at

To our knowledge, this is the first longitudinal and prospective study involving Brazilian chil- dren that demonstrated the practice of NEBF to be associated with an increased risk

While Barrett’s esophagus develops as a result of chronic gastroesophageal refl ux disease, intestinal metaplasia in the gastric cardia is a consequence of chronic Helicobacter

Conclusion - Due to the low prevalence of Barrett’s esophagus found in the present study, associated with the finding of short-segment Barrett’s esophagus in all cases diagnosed

Results - The analysis of the dietary pattern of functional dyspeptic patients with intestinal metaplasia, and its comparison with those without intestinal metaplasia, showed

These indings disagree with the previous report in the same area that identiied 29% of patients underwent upper GI endoscopy presenting intestinal metaplasia in the distal