• Nenhum resultado encontrado

Targeting cancer-related inflammation: Chinese herbal medicine inhibits epithelial-to-mesenchymal transition in pancreatic cancer.

N/A
N/A
Protected

Academic year: 2017

Share "Targeting cancer-related inflammation: Chinese herbal medicine inhibits epithelial-to-mesenchymal transition in pancreatic cancer."

Copied!
9
0
0

Texto

Loading

Imagem

Figure 2. QYHJ inhibits the growth of human pancreatic tumors in an orthotopic nude mouse model
Figure 4. QYHJ suppresses cancer-related inflammation in pancreatic cancer. A. An orthotopic nude mouse model was established and treated with QYHJ or vehicle as described above
Figure S1 The fingerprint chromatograms of QYHJ formula. A. High-performance liquid chromatography (HPLC)

Referências

Documentos relacionados

There is ample evidence of the importance of E-cadhe- rins to inhibit tumor invasion and metastases: the metasta- tic potential of various cancer cell lines is inversely related to

In the present study, we have developed a spheroid-based, 3-D culture of pancreatic cancer cell lines MIAPaCa-2 and PANC-1 for pancreatic drug testing, using the acid

Using pancreatic cancer cell lines with or without expressing mutated K-ras , we demonstrated that the inhibition of endogenous PKC activity sensitized human pancreatic cancer

Forced expression of miR-7 in aggressive breast cancer cell lines suppressed tumor cell monolayer proliferation, anchorage independent growth, three-dimensional growth in

In this study, we have shown that soluble factors secreted by activated PBMC of healthy donors including TNF- α , IL-6 and TGF- β can induce a paradoxical co-expression of EMT

In the present study, we addressed the heterogeneity in pancreatic cancer cell lines BxPC3 [22] and PC3 [23] based on the morphology of colonies derived from single cancer cells

Heterogeneity tests indicated that studies analyzing the association between nine cancers (EC, gastric cancer, lung cancer, HCC, colorectal cancer, pancreatic cancer, female

These results indicate that miR-200c overexpression regulates MUC4 and MUC16 mucins in pancreatic cancer cells by directly targeting the mRNA coding sequence of