Exposure to ozone modulates human airway protease/antiprotease balance contributing to increased influenza A infection.

Silencing of FANCF in breast cancer cell lines led to significantly increased DNA damage compared with the cells treated with control shRNA (Figure 3).. Silencing of FANCF induced

We demonstrate that treatment of human embryonic kidney cells, alveolar epithelial cells, human breast cancer cells or colon cancer cells with heat, or TRPV1 agonists, capsaicin or

Cultures of human A549 cells were transfected with SFPQ/PSF- specific (S1) or control siRNAs (siCTRL) as described under Materials and Methods and then infected with influenza virus

A consolidação dos dados foi realizada com a finalidade de se obter os seguintes índices: coeficiente geral de detecção de casos novos de hanseníase por 100 mil

Furthermore, we extended our study to include insulin-producing cells differentiated from human induced pluripotent stem (iPS) cells to establish a viral infection model of

Increased expression of Rev3 in human breast epithelial cancer cell lines compared to non- neoplastic human breast epithelial cells MCF-12A, and in human breast tumors compared

This endobronchial spread requires apical infection of differentiated human bronchial epithelial cells and apical release of infectious virus progeny as demonstrated for HAdV-B14p1

Here we established primary culture of undifferentiated and well differentiated normal human bronchial epithelial (NHBE) cells and infected with highly pathogenic influenza H5N1