Impaired bone formation in Pdia3 deficient mice.

Thus, the results suggest that both bone surface and whole bone are suitable biomarkers of chronic F exposure in rats and plasma may be used as indicator of bone fluoride

Thus, we examined the effects of osteocyte apoptosis and the reductions in the numbers of osteocytes and their processes on bone formation and resorption using BCL2 transgenic mice at

Skeletal analyses of Gli1 + / 2 adult mice revealed that Gli1 haploinsufficiency caused decreased bone mass with reduced bone formation and accelerated bone resorption, suggesting

mRNA levels of Trx1 , Sod2 , Prdx3 and Gpx1 in the skeletal muscle of wild type (WT) and dyslipidemic (ATX) mice from the sedentary (SED) and exercise (EX) groups.. Trx1: thioredoxin

Partial nephrectomy induced the hepatic ALAS1 in both wild type and AIP mice. The up-regulated heme biosynthesis in the AIP mice with genetically deficient PBGD activity gave rise to

We found that CD55-deficient mice, like mice that lack CD97, had increased levels of circulating granulocytes, which was due to a higher granulopoietic activity in the bone

In the present study, we evaluated serum levels of bone-specific alkaline phosphatase (BALP), bone formation marker, and carboxyterminal telopeptide of type I collagen (CTX-I),

Our fi ndings demonstrated that SR enhanced bone formation and inhibited bone resorption in a wear particle-mediated osteolysis model in wild-type mice, and this effect relied mainly