Imunoexpressão de Rankl, CCL5/RANTES e CCR5 em inflamação pulpar aguda em ratos, induzida por exposição tecidual, ácido lipoteicóico ou lipopolissacarídeo

Neste trabalho analisamos por citometria de fluxo, níveis séricos de cinco quimiocinas (IL-8/CXCL8, MCP-1/CCL2, MIG/CXCL9, IP-10/CXCL10 e RANTES/CCL5) e nove citocinas (IL-1

Receptor activator of nuclear factor kappa B ligand (RANKL) expres- sion is induced in osteoblasts, activated T-cells, synovial fibroblasts and bone marrow stromal cells, and

Total phenolic, fl avonoid, alkaloid and iridoid content and preventive effect of Lider-7-tang on lipopolysaccharide-induced acute lung injury in

In this study after a single systemic LPS injection the expression of TNF- a in the serum preceded the up-regulation of IL-1 b (Figure 1B) suggesting that activation of the

Despite the buzz generated by the potential ‘external’ tissue engineering (and tooth-engineering) application of dental pulp stem cells, its ‘intrinsic’ role in dental pulp and

LPS, lipopolysaccharide; MP, methylprednisolone; LM, LPS groups treated with MP; iNOS, inducible nitric oxide synthase; AGEs, advanced glycation end products; RAGE, receptor for

Course of infection, chemokine expression, and protein production at the site of infection in IL-12, IFN- ␥ , and IL-4 knockout ( ⫺ / ⫺ ) mice and their wild-type control..

In the glucocorti- coid-induced osteoporosis the RANK/RANKL/ /OPG role was described: glucocorticoids stimu- late RANKL expression by osteoclasts and inhibit OPG synthesis,