Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update

killing, as the parasite is too big (15) to be ingested by the macrophage. Therefore, in an in vivo infection, activated macrophages should use an easily diffusible chemical spe-

In endothelial cells and cardiac myocytes eNOS is localized in specialized plasmalemmal signal-transducing domains termed caveolae; acyla- tion of the enzyme by the fatty

endotoxin induces the release of endothelium-derived nitric oxide (EDNO), an endogenous nitrovasodilator, segments of canine femo- ral, renal, hepatic, superior mesenteric, and

Nitric oxide synthase (NOS) inhibi- tion by L-NAME blocked vascular relaxation and reduced superoxide production in LPS-treated animals.. In the presence of L-NAME there was no

Endocrine and metabolic disorders have been variably reported to be associated with human T-cell leukemia virus type 1 (HTLV-1) infection.. Therefore, the aim of this article was

Takeuchi, Role of nitric oxide in regulation of gastric acid secretion in rats: effects of NO donors and NO synthase inhibitor, Br. Kim, Effects of a nitric oxide donor and nitric

The present study evaluated the effect of alterations in the levels of nitric oxide (NO) and superoxide anion in the caudal ventrolateral medulla (CVLM), a key area of the brainstem

We observed the induction of cell death and the production of nitric oxide, hydrogen peroxide, interleukin-8 and vascular endothelial growth factor A in human umbilical