Stress-induced PARP activation mediates recruitment of Drosophila Mi-2 to promote heat shock gene expression.

rates in genes in relation to the core genes in the complete genome and codon bias (as in surrogate methods) are taken into account, a specific model of horizontal transfer can

Pim-1 kinase inhibits the activation of reporter gene expression in Elk-1 and c-Fos reporting systems but not the endogenous gene expression: an artifact of the reporter gene assay

Notably, we identify the transcription factor Efg1p to be required for expression in response to heat shock and oxidative stress and demonstrate that expression of Ca Hsp12p

We found that Med19 activity is required in the Drosophila haltere disc for transcriptional activation of CG13222 / edge and bab2 , but is dispensable for Ubx-mediated repression

Using in vivo gene transfer we show that LXR activation by its synthetic agonist GW3965 represses the transcriptional activity of two key metabolic genes, Thyrotropin-releasing

In this, the first comprehensive analysis of gene expression changes in response to heat shock and MG132 (a proteasome inhibitor), both of which are known to induce heat shock

Expression and splicing of the insulin-like growth factor gene in rodent muscle is associated with muscle satellite (stem) cell activation following local

Following transcription, post-transcriptional and post-translational mechanisms of regulation of gene expression have also been shown to play an important role in