Experimental Hepatic Fibrosis Due to Capillaria hepatica Infection (Differential Features Presented by Rats and Mice)

For the study of hepatic metabolism, microsomal fractions isolated from liver of Wistar rats and Balb-C mice were used, with protein concentrations of 30 to. 40 mg

Studies performed with rats and mice inoculated orally with Trypanosoma cruzi, obtained from blood of infected mice or infected feces from triatomid bugs, showed that the

gondii, where the knockout mice were more resistant to acute infection compared to WT mice and presented little evi- dence of inflammatory changes in the liver and small intestine

2 - Box and whisker plots representing the numbers of spleen cells from mice control and at days post-infection (dpi) by Dermatobia hominis submitted to RPMI... 3 - Box and

cruzi - infected mice at 14 and 28 days post-infection, 400X; B: cytofluorimetric analysis of mononuclear cells enzymatically harvested from cardiac tissue showed that at 28 dpi most

Systhematized septal fibrosis of the liver can be induced in rats either by repeated intraperitoneal injections of pig-serum or by Capillaria hepatica infection.. The

3: mortality rate of the three strains in mice was evaluated from seven to 48 days after infection and was expressed as percent- age of dead animals, varying from a low (5 to 10%)

BALB/c mice were inoculated with 5-500 Toxocara canis infective eggs, and bled at 15-120 days post infection (dpi) to evaluate the dynamics of IgG antibody response and