TCDD-Induced Activation of Aryl Hydrocarbon Receptor Inhibits Th17 Polarization and Regulates Non-Eosinophilic Airway Inflammation in Asthma.

In the process of apoptosis, cell death is induced by the activation of a genetically and biochemically regulated cell death system, involving the participation of

As mast cell activation has been shown to be a source of induction of eosinophil infil- tration as well as AHR (10,22), it is conceiv- able that mast cells play an important role in

In conclusion, we found that endothelial cells were mediated in the abnormal differentiation of Th subsets, and adiponectin inhibited the differentiation of Th1 and Th17 subsets

The expression pattern and nuclear localization suggest that Mipu1 plays a role in the regulation of gene transcription in the cardiovascular system.. Upregulation of Mipu1 is

Because differential activation of canonical Wnt signaling in FOb and POb plays an important role in determining substantial differences in their osteogenic potential, as well as

Since the Ahr gene, besides its role in the regulation of morphogenesis, also plays a role in cellular reaction to toxin exposure, and, in particular, to ionizing radiation [18],

When viewed together, epicutaneous sensitization of wild type mice with superantigen SEB enhanced epicutaneous Ova-induced lung inflammation, airway mucus metaplasia, AHR and Th2

As Th17 cells play a central role in pathogenesis of RA and VSTM1-v2 is involved in Th17 cell differentiation, we further detected RNA expression of IL-17A, and analyzed