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Age-dependent neuroplasticity mechanisms in Alzheimer Tg2576 mice following modulation of brain amyloid-β levels.

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Academic year: 2017

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Figure 1. Effects of (+)-phenserine on brain amyloid levels in Tg2576 mice. Levels of tris-soluble (soluble) and guanidine-soluble (insoluble) Ab1-42 and Ab1-40 in the cerebral cortices of 4- to 6-month-old (A) and 15- to 18-month-old (B) Tg2576 transgenic
Figure 2. Modulation of amyloid-b (Ab) burden in 15- to 18-month-old Tg2576 mice treated with (+)-phenserine
Figure 7. Correlations of amyloid-b (Ab) levels with synaptophysin levels, cell proliferation and proinflammatory markers in the brains of 4- to 6- and 15- to 18-month-old Tg2576 mice

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