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Modulation of ocular surface glycocalyx barrier function by a galectin-3 N-terminal deletion mutant and membrane-anchored synthetic glycopolymers.

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Academic year: 2017

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Figure 2. Abrogation of galectin-3 impairs barrier function in mouse corneas. Numerical scoring for the intensity of staining with rose bengal revealed a higher incidence of epithelial defects in corneas of galectin-3 null mice (Gal-3 2/2 ) as compared to
Figure 3. Disruption of galectin-3 binding and multimerization impairs glycocalyx barrier function
Figure 5. Cellobiose glycopolymers impair glycocalyx barrier function in human corneal epithelial cells

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