Immunobiological responses to short carbohydrate epitopes in Trypanosoma cruzi

Mice with disrupted IL-10 genes (IL-10 KO) or with disrupted recombinase-activat- ing genes that completely lack T and B cells (RAG/KO) provide a model to investigate the role of

At the chronic phase of infection, parasite replication is counteracted by parasite killing mediated by cytotoxic CD8 + T-cell responses (indicated in Figure 1, top.. left), and

Enlarged map shows the spatial distribution of neighborhood dogs according to blood test results, in relation to Leishmania spp., Toxoplasma gondii , Trypanosoma cruzi and

There is not change on intracellular basal calcium level; B: control done without parasite addition, showing the liquid turbulence effect as a transient calcium signal during PBS

In- cubation of membranes with plasminogen and ε - aminocaproic acid abolished the binding to the parasite proteins, indicating specific binding through lysine bind- ing sites in

The slightly inhibitory effect of very high concentra- tions of NAOx or NPOx (10 mM) on glucose-6-phosphate dehydrogenase, HADH-isozyme and malic enzyme, and the lack of any

The differential regulation mechanisms of the iNOS gene in macrophages and glial cells (Gazzinelli et al. 1998, Saha & Pahan 2006) could explain the greater infection

While no differences in the magnitude of T-cell responses specific for KMP-11 and HSP-70-derived peptides according to the clinical stage of the disease were found, IFN-γ