• Nenhum resultado encontrado

Kaempferol inhibited VEGF and PGF expression and in vitro angiogenesis of HRECs under diabetic-like environment

N/A
N/A
Protected

Academic year: 2019

Share "Kaempferol inhibited VEGF and PGF expression and in vitro angiogenesis of HRECs under diabetic-like environment"

Copied!
7
0
0

Texto

Loading

Imagem

Figure 1. Effect of high glucose (25 mM) for 12, 24, or 48 h on mRNA expression levels of (A) vascular endothelial growth factor (VEGF) and (C) placenta growth factor (PGF) expression in human retinal endothelial cells (HRECs measured by qRT-PCR
Figure 4. Effects of kaempferol on high glucose- glucose-induced PI3K expression and Erk1/2, Src, and AKT1 activation

Referências

Documentos relacionados

The main objectives of this study were to determine whether DNA hypomethylation correlated with vascular endothelial growth factor-C (VEGF-C) expression, and the effect of SAM on

The death of retinal ganglion cells (RGC) was evaluated in histological sections, and retinal ACE2, caspase-3, and vascular endothelial growth factor (VEGF) expressions were analyzed

The main regulating factors of the luteal angiogenesis are: vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF) and angiopoetins, which promote

A, Vascular endothelial growth factor (VEGF) allows the transformation of endothelial cells into pericytes or smooth muscle cells and increase the number of pericytes

As cefaleias tipo tensão (60,8%), a enxaqueca (24,8%) e as ce- faleias atribuídas a perturbações psiquiátricas (7,2%), foram os tipos mais prevalentes, enquanto as perturbações

Plasma production and cardiac expression of the vascular endothelial growth factor (VEGF) in Trypanosoma cruzi infected animals.. The VEGF was measured through ELISA in (A) the

2: expression of vascular endothelial growth factor (VEGF) receptors during human dendritic cell differentiation and subsequent matura - tion in the presence of prostaglandin E2

Objective: This article aimed at identifying the expression of fibroblast growth factor-2 (FGF-2) and vascular endothelial growth factor (VEGF) in the tension and pressure areas of