• Nenhum resultado encontrado

A physiology-based approach to a patient with hyperkalemic renal tubular acidosis Abordagem diagnóstica de um paciente com acidose tubular renal hipercalêmica

N/A
N/A
Protected

Academic year: 2019

Share "A physiology-based approach to a patient with hyperkalemic renal tubular acidosis Abordagem diagnóstica de um paciente com acidose tubular renal hipercalêmica"

Copied!
8
0
0

Texto

Loading

Imagem

Figure 1. (A) Pretreatment electrocardiogram with peaked T-waves,  flattening of the P-wave, prolonged PR interval, and widening  of the QRS complex
Figure 2. Interaction between potassium and proton excretion and ammoniagenesis. Sodium reabsorption by ENAC transporter in principal cells,  driven by Na + /K + -ATPase, creates a lumen-negative transepithelial voltage that is critical for potassium (by R
Figure 3. Pathophysiologic classification and etiologies of disorders associated with hyperkalemic hyperchloremic renal tubular acidosis
Figure 4. Clinical approach to the diagnosis of hyperkalemic RTA based on urine pH. Adapted from reference 1

Referências

Documentos relacionados

Além disso, é importante enfatizar que a TFG está relati- vamente preservada nos diferentes tipos de ATR, e o grau de disfunção renal encontrado no presente caso não pode

nephrology clinic since 1995 due to recurrent nephroli- thiasis and medullary nephrocalcinosis resulting from renal tubular acidosis (urinary pH of 8 in the context of systemic

Although renal replacement therapy is rarely needed in rhabdomyolysis, it should be considered when there is severe and resistant hyperkalemia, persistent metabolic acidosis,

Metabolic acidosis in adults may be caused by increased acid generation (as in ketoacidosis and in lactic acidosis), by loss of bicarbonate (diarrhea o r type 2 renal tubular acidosis

Metabolic acidosis caused a sustained increase in renal fractional so- dium excretion and potassium excretion which was accompanied by a rise in the fractional proximal

It is described that the increase in sodium and water loss from the collecting duct leads to a reactive increase in sodium and water reabsorption by the proximal tubule [5], but even

The results of this cross-sectional analysis of renal function in hepatosplenic schistosomiasis clarify important aspects of tubular dysfunction in these patients and demonstrate

Genetic investigation of autosomal recessive distal renal tubular acidosis: evidence for early sensorineural hearing loss associated with mutations in the ATP6V0A4 gene. Karet