The loss of miR-26a-mediated post-transcriptional regulation of cyclin E2 in pancreatic cancer cell proliferation and decreased patient survival.

Forced expression of miR-7 in aggressive breast cancer cell lines suppressed tumor cell monolayer proliferation, anchorage independent growth, three-dimensional growth in

So, does this mean that if we block the activity of caspases we will prevent every sign of apoptosis and therefore keep the cells alive? The answer is yes in two situations: a)

Additionally, we compared the anticancer effects of gemcitabine and berberine by evaluating cellular growth, cell cycle, and apoptosis in two pancreatic cancer cell lines.. Material

The probability of attending school four our group of interest in this region increased by 6.5 percentage points after the expansion of the Bolsa Família program in 2007 and

The effect caused by the TLC purified yellow pigment on the growth and proliferation of Human cervical cancer cell line (HeLa) and Human liver cancer cell line

To analyse the expression of miR-196a, qRT-PCR was performed in Four human pancreatic cancer cell lines (PANC-1, Capan-2, BxPC-3 and SW1990) and an immortalized pancreatic

To study the effects of miR-125a down-regulation on pro- liferation and invasion of HCC, lentivirus-mediated siRNA targeting the precursor of miR-125a was transfected into

Our goal is to retain the elegance of the Boolean representation of the switching network, while introducing continuous variables for cell size, cell age, and cyclin composition,