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Arrest of nuclear division in Plasmodium through blockage of erythrocyte surface exposed ribosomal protein P2.

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Figure 3. Immunoblot, mass spectrometry, flow cytometry and IFA of parasite and RBC localized PfP2 protein
Figure 5. Effect of anti-P2 mAb E2G12 on Plasmodium cells. Synchronized P. falciparum cells were treated with no antibody (control); SP2/O:
Figure 6. BODIPY-ceramide and FM4-64 staining of live infected erythrocytes in the presence and absence of anti-P2 mAb E2G12.

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