• Nenhum resultado encontrado

Joint effect of genetic and lifestyle risk factors on type 2 diabetes risk among Chinese men and women.

N/A
N/A
Protected

Academic year: 2017

Share "Joint effect of genetic and lifestyle risk factors on type 2 diabetes risk among Chinese men and women."

Copied!
7
0
0

Texto

Loading

Imagem

Table 5 shows joint effects between BMI, WHR, exercise participation and lifestyle combined risk factors with GRS2 (tertiles)
Table 4. Joint Effects of BMI, WHR, exercise participation and combined lifestyle risk factor with GRS1categories on T2D.
Table 5. Joint Effects of BMI, WHR, exercise participation and combined lifestyle risk factor with GRS2 categories on T2D.

Referências

Documentos relacionados

For instance, data from the UK Health and Lifestyle Survey investigating the same lifestyle risk factors as in the current study found similar levels of risk for all- cause mortality

We conducted a genome-wide association study (GWAS) in a homogenous case-control cohort from Bergen, Norway (823 COPD cases and 810 smoking controls) and evaluated the top 100

In this study, genome-wide association analysis (GWAS) was exploited to detect the quantitative trait loci (QTL) for num- ber of days to flowering (ETF), number of days from

Joint effects of CPE and IDE genetic polymorphisms We tested the joint effects of rs1583645 in CPE and rs6583813 in IDE on T2D risk in the Asian case-control cohort and beta

We evaluated the socioeconomic aspects, body composition, risk of metabolic complications associated with obesity, eating habits and lifestyle in both women and men adults and

The purpose of this study was to extend analysis of data from a previous genome-wide association study (GWAS) of T2D-ESKD in the African American population [5] with detailed

candidate region SNPs (chosen mostly from recent GWAS) and performed a genome-wide admixture mapping scan aimed at detecting loci carrying Native American T2D susceptibility alleles

Genome-wide association studies (GWAS) of CRC have con- firmed the hypothesis that part of the heritable risk for this disease is caused by common, low-risk variants and have