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Myenteric plexus is differentially affected by infection with distinct Trypanosoma cruzi strains in Beagle dogs

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Academic year: 2019

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Fig. 1: tissue parasitism and inflammatory process evaluation in the oesophagus and colon of Beagle dogs experimentally infected with Y or  Berenice-78 (Be-78) Trypanosoma cruzi strains
Fig. 2: quantitative evaluation of total soluble collagen in the oesophagus and colon of Beagle dogs necropsied in acute (□) or chronic (■)  phases from experimental infection with Y or Berenice-78 (Be-78) strains of Trypanosoma cruzi
Fig. 3: morphometric analysis of Auerbach’s plexus PGP9.5-immunoreactive (IR) neurons and nerve fibres (A) and glial fibrillary acidic pro- pro-tein (GFAP)-IR enteroglial components (B) in the oesophagus and colon of Beagle dogs necropsied in acute (□) or
Fig. 4: photomicrographs of histological sections of the oesophagus showing Auerbach’s plexus PGP9.5-IR neurons and nerve fibres and glial  fibrillary acidic protein-immunoreactive (GFAP-IR) enteroglial components in noninfected (NI) Beagle dogs or dogs in

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