C/EBPβ-LAP*/LAP Expression Is Mediated by RSK/eIF4B-Dependent Signalling and Boosted by Increased Protein Stability in Models of Monocytic Differentiation.
Texto
Imagem
Documentos relacionados
immunofluorescence microscopy.. Phagocytosis by THP-1 cells is antibody and Fc Receptor dependent. A) EtBr stained merozoites were incubated with i) non- immune plasma or ii) immune
PBMC were stimulated with BRSV for 24 hours and then analyzed for expression of IL-17 by qPCR (C); or for 6 days and then cell culture supernatants were analyzed by ELISA for
Cells stably expressing the control shRNA or Snail1 shRNA were treated with TGF- b 1 (3 g/ml) for 6 days and expression of E-cadherin and N-cadherin were determined by qRT-PCR (E)
In the present study, BPE treatment strongly suppressed C/EBP b mRNA and protein expression and markedly reduced the expression levels of C/EBP a and PPAR c compared with those
( d ) P21 mRNA and protein levels were upregulated in ECC-1 cells treated with TSA for 24 h, while it was partially neutralized by miR-106b (left). BIM mRNA was upregulated in
siRNAs directed against (A) luciferase or XIAP, (B) cIAP-1, (C) cIAP-2, or (D) survivin were transfected in HeLa cells, and the protein levels of all IAPs were determined in infected
GARP expression correlated with surface LAP expression on both Foxp3 + cells and Foxp3 2 cells (Figure 4B), suggesting that surface LAP is GARP-dependent not only on natural Tregs
AM attenuated cell cycle proliferation arrest of TGFß on HaCaT cells and induced the expression of c-Jun protein in HaCaT and in human primary keratinocytes.. Treatment of HaCaT