Specific humoral immunity versus polyclonal B cell activation in Trypanosoma cruzi infection of susceptible and resistant mice.

Since many factors may be involved, we organized a systematic study of the effects of aging on the immune system of rodents, simulta- neously considering the main aspects of

One plausible explanation [6] for the absence of systemic cytokine release in CD28SA treated mice could be instantaneous consumption of IL-2 by Treg cells, which would contribute

In susceptible BALB/c mice and resistant C57BL/6 mice that were intradermally inoculated with a low dose of Leishmania major in the ear, we investigated the vari- ation in

Nitric oxide- induced apoptotic cell death in the acute phase of Trypanosoma cruzi infection in mice.. S-nitrosylation regulates

6: Western blot analysis of levels of Trypanosoma cruzi ascorbate peroxidase (TcAPX) protein in benznidazole BZ-susceptible and BZ- resistant T.. cruzi populations

We investigated the resistance to Strongyloides venezuelensis primary infection of mice strains NIH (resistant) and C57BL/6 (susceptible) and the F1 and F2 offspring of crosses

aureus and the course of pneumonia was analyzed in comparison with uninfected mice regarding clinical signs, bacterial clearance, leukocyte immigration and cytokine response

To investigate CII antigen-specific T cell response and cytokine production in CIA wild type and B7-H3 KO mice, splenocytes were removed from arthritic mice 20 days after the