Schisandrin B prevents doxorubicin induced cardiac dysfunction by modulation of DNA damage, oxidative stress and inflammation through inhibition of MAPK/p53 signaling.
Texto
Documentos relacionados
Oxidative stress, cytokine/chemokine and disruption of blood-brain barrier in neonate rats after meningitis by Streptococcus agalactiae. A kinetic study of the cytokine/chemokines
Higher concentrations of plasma MMP-8 and MMP-9 were measured in asymptomatic patients in hydrocortisone group at 1 hour and 6 hours after CEA, while lower serum levels of MMP-8
For the study of resistance to reinfection induced by selected irradiation dose and cytokine production, the experiment groups were designed as follows: uninfected control,
Our results showed that psychological stress induced high levels of corticosterone and ACTH in plasma and increases in proinflammatory cytokines IL-1β and TNF-α in a
The results indicated that treadmill training had a protective effect against damage and dysfunction, attenuated the increase in DOX-induced apoptosis, and increased
Expression of hMTH1 in the transgenic mice conferred a significant protection against paraquat-induced oxidative DNA damage and DNA 8-oxodG levels in these organs were between 4-
To investigate CII antigen-specific T cell response and cytokine production in CIA wild type and B7-H3 KO mice, splenocytes were removed from arthritic mice 20 days after the
The results of this study suggest that inflammation-induced BM exhaustion and a disturbed progenitor cell mobilization response due to reduced levels and activity of MMP-9 in the BM