Human immunodeficiency virus impairs reverse cholesterol transport from macrophages.

Objective: To determine the prevalence and the risk factors for Human Immunodeficiency Virus (HIV) Infection in pregnant women admitted to the President Prudent State Hospital,

One hundred clinical isolates of Cryptococcus neoformans from human immunodeficiency virus (HIV)- infected and non-HIV-infected patients from Brazil, Chile, and Venezuela were

To assess the prevalence of primary resistance of human immunodeficiency virus type 1 (HIV-1) to antiretrovirals, 84 patients chronically infected with HIV without prior

Women with human immunodeficiency virus (HIV) infection present a higher risk of infection by the human papil- lomavirus (HPV) and cervical cancer.. To determine HPV genotypes

In order to estimate the prevalence of human immunodeficiency virus type 1 (HIV-1) and hepatitis C virus (HCV) co-infection in hard-to-reach intravenous drug users, 199 subjects

Abstract The purpose of this study was to determine the seroprevalence of human immunodeficiency virus (HIV-1/2), human T-cell lymphotropic virus (HTLV-I/II), hepatitis B virus

To determine the role of PDE8A in HIV-1 replication in macrophages, we analyzed the effect of PDE8A knockdown by shRNAs on the HIV-1 replication cycle in these cells.. We selected

However, considering that the reverse cholesterol transport is a protective pathway of lipoprotein metabolism against cholesterol accumulation in the body, and considering that