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Atorvastatin therapy during the peri-infarct period attenuates left ventricular dysfunction and remodeling after myocardial infarction.

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Academic year: 2017

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Figure 2. Echocardiographic assessment of LV function. Representative two-dimensional (A, C) and M-mode (B, D) images from vehicle- vehicle-treated (A, B), and atorvastatin-vehicle-treated (C, D) rats 4 wk after coronary occlusion
Figure 3. Hemodynamic assessment of LV function at 4 wk after atorvastatin treatment. Representative pressure-volume loops from a vehicle-treated (A) and an atorvastatin-treated (B) rat recorded during preload manipulation by a brief period of inferior ven
Figure 4. Quantitative assessment of LV remodeling. Panels A–D illustrates echocardiographic measurement of LV AW thickness in diastole (A), LV end-diastolic diameter (B), LV end-diastolic area (C) and LV end-diastolic volume (D) at baseline, 48 hr and 4 w
Figure 6. Myocardial collagen content. Panels A–B shows the quantitative assessment of collagen content in the ischemic zone (A) and non-ischemic zone (B) at 4 wk after myocardial infarction

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