Regulation of Adipogenesis and Key Adipogenic Gene Expression by 1, 25-Dihydroxyvitamin D in 3T3-L1 Cells.

Tumor necrosis factor-alpha-induced insulin resistance in 3T3-L1 adipocytes is accompanied by a loss of insulin receptor substrate-1 and GLUT4 expression without a loss

Application of mechanical strain to cells cultured in adipogenic medium significantly limited the rise in C/EBPb mRNA expression which occurred in early adipogenesis, compared

Up-regulation of X-linked gene expres- sion in male and female cells is consistent with the overall ; 1:1 expression ratio of X-linked and autosomal genes calculated from analysis

Regulation of herpes simplex virus type 1 replication in Vero cells by Psychotria serpens : relationship to gene expression, DNA replication, and protein

Figure 2 - JMJD2C expression in the 3T3-L1 cell line, capacity of binding to histone deacetylases and – to PPAR g (A) RNA extractions and RT-PCR assays for JMJD2c were performed

After radiation, the high expression of mPer2 in NIH 3T3 cells results in reduced cell death and enhanced cell proliferation and clonogenic survival, which means that

regulates the ability of 3T3-L1 cells to undergo adipocyte differentiation by preventing the expression of critical adipogenic transcription factors, such as C/ EBP β / δ , SREBP1

Fur- thermore, mRNA levels in undifferentiated 3T3-L1 cells remained high in response to lipotoxic challenges even after drop-offs in NF- kB and AP-1 activity between 4 and 8