Involvement of miR-30c in resistance to doxorubicin by regulating YWHAZ in breast cancer cells

Forced expression of miR-7 in aggressive breast cancer cell lines suppressed tumor cell monolayer proliferation, anchorage independent growth, three-dimensional growth in

In the present study, we provide evidence that miR-29c expression in high-metastatic 95D cell lines was downregulated when compared to miR-29c expression in paired low-metastatic

In this study, we found that miR-26a was strongly downregulated in gastric cancer (GC) tissues and cell lines, and its expression levels were associated with lymph node metastasis

Increased expression of Rev3 in human breast epithelial cancer cell lines compared to non- neoplastic human breast epithelial cells MCF-12A, and in human breast tumors compared

IL-1ß downregulates the expression of MITF-M and upregulates the expression of miR-155 in 2 melanoma cell lines.. The result was normalized to ß-actin expression (means ± SD for 3 or

To overcome this hurdle for studying the functional role of Vav1 in human breast cancer, we overexpressed Vav1 in two breast cancer cell lines, AU565 and MCF-7, achieving Vav1

In the current study, on a tissue level, miR-138 expression was not significantly changed in breast tumour samples compared to healthy controls, but was found to be

(A) miR-320a expression was decreased in SW480/ shE2A cells as shown by qRT-PCR; (B) Co-transfection of E12 or E47 into SW480/shE2A cells could normalize the expression of miR-320a;