Rev. Bras. . vol.72 número1 en a07v72n1

The loss of GM-CSF activity caused by anti-GM-CSF antibodies may impair the normal function of pulmonary alveolar macrophages and therefore decrease the surfactant clearance

Objectives The aim of this investigation was to assess the concentration of GM-CSF in nasal secretions of patients with non-allergic rhinitis with eosinophilia syndrome (NARES)

Co-administration of pGM-CSF enhances HIVBr18- induced T-cell responses - In order to evaluate whether GM-CSF would enhance HIVBr18 DNA vaccine im- munogenicity, we

We present the results of the first clinical trial that evaluated the safety of rhu-GM-CSF as adjuvant immunotherapy for the treatment of active human pulmonary tuberculosis..

In the presence of GM-CSF plus IL-4 for 7 days is observed the differentiation of CD14 + cells into immature DCs as evaluated by FACS with a significantly higher expression of CD1a

Among these growth factors, the evaluation of M-CSF and GM-CSF production by macrophages is not just important to understand the immune response, but also to understand

SUMMARY — W e detected the cytokines interleukin-6 (IL-6) and granulocyte macrophage- CSF (GM-CSF) by ELISA in the CSF and serum of 30 HIV-infected patients classified as

The small number of abnormal values for CSF limits the ability of the current findings to confidently assess sensitivity and positive predictive values, however, our results