Rev. Bras. . vol.72 número1 en a07v72n1

The loss of GM-CSF activity caused by anti-GM-CSF antibodies may impair the normal function of pulmonary alveolar macrophages and therefore decrease the surfactant clearance

Objectives The aim of this investigation was to assess the concentration of GM-CSF in nasal secretions of patients with non-allergic rhinitis with eosinophilia syndrome (NARES)

Among these growth factors, the evaluation of M-CSF and GM-CSF production by macrophages is not just important to understand the immune response, but also to understand

SUMMARY — W e detected the cytokines interleukin-6 (IL-6) and granulocyte macrophage- CSF (GM-CSF) by ELISA in the CSF and serum of 30 HIV-infected patients classified as

The small number of abnormal values for CSF limits the ability of the current findings to confidently assess sensitivity and positive predictive values, however, our results

In the presence of GM-CSF plus IL-4 for 7 days is observed the differentiation of CD14 + cells into immature DCs as evaluated by FACS with a significantly higher expression of CD1a

Co-administration of pGM-CSF enhances HIVBr18- induced T-cell responses - In order to evaluate whether GM-CSF would enhance HIVBr18 DNA vaccine im- munogenicity, we

We present the results of the first clinical trial that evaluated the safety of rhu-GM-CSF as adjuvant immunotherapy for the treatment of active human pulmonary tuberculosis..