High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

To investigate the differential gene expression in bladder carcinoma compared to normal cells and the possi- ble mechanisms involved in the initiation of bladder cancer, we analyzed

nels (VGSC) in the progression of different tumors, such as prostate cancer, small cell lung cancer, breast cancer and others, linking VGSC to the invasion capacity of tumor cells (4

Normal and tumor cells progress through the cell cycle in similar ways, but the higher proliferation rates of cancer cells cause mitotic targets to be more frequently expressed

Our model illustrates how interactions between the genotype of normal and neoplastic cell populations, combined with the individual host specificities, determine both the phenotype

Therefore, to determine the relationship between TP53 status and the tumor response to chemotherapeutic agents, we analyzed the genotoxic effect of cisplatin and gemcitabine, and

We inves- tigated the expression of inflammatory cells and cytokines in the liver and serum of 51 chronically HCV infected patients and compared them to data from two sets of

Roche a igualizar o recinto de buracos de poste a vestígios de uma cabana, da mesma forma que fossas e lareiras foram lidas como vestígios de áreas de habitat; neste

was used to analyze the differences in the expression of active and total Src in cytoplasm and membrane between normal ducts and cancer tissue, between African American and