Salinomycin enhances doxorubicin sensitivity through reversing the epithelial-mesenchymal transition of cholangiocarcinoma cells by regulating ARK5

In HNSCC, aberrant HGF/c-MET signaling is involved in tumor progression by promoting EMT (Epithelial Mesenchymal Transition), cell migration, invasion, proliferation

Diana Micro-T Target prediction made with miRNA or mRNA sequences as input diana.cslab.ece.ntua.gr/ microT/ FAME Experimentally verified miRNA pathways infer biological

To further confirm that the UA-mediated inhibition of cell proliferation is through regulation of COX-2 signaling in colon cancer cells, we pretreated SW480 cells with celecoxib (CB,

The scores obtained after the application of PRUNET to four biological examples, namely epithelial to mesenchymal transition (EMT), Th1-Th2 transdifferentiation (Th1-Th2),

We conclude that 1) acidosis induces epithelial-to- mesenchymal transition and secretion of TGF- β 1 to the cellular supernatant of HK2 cells; and 2) that ci- trate improves

In order to approach the problem of EMT in cholangiocarcinoma, we decided to investigate the changes in protein expression occurring in two cell lines under conditions leading to

Using cholangiocarcinoma cells as a model of TRAIL resistance, we found that Hedgehog signaling blockade sensitized these cancer cells to TRAIL cytotoxicity independent of

indeed promotes EMT in these ovarian cancer cells, we examined the expression of E-cadherin and N-cadherin, two well-established epithelial to mesenchymal makers, in