Split-cre complementation indicates coincident activity of different genes in vivo.

To assess the effectiveness of the model we compared the generated solutions with the solutions generated via two other different methods: (1) a portfolio split

Cis -complementation of essential MCMV genes in cells In order to test the basic principle, Flpe-NIH were nucleofected with MCMV BACs which were generated by deletion of the

To investigate the mechanism of lymphoma regression, we tested whether Cre activation could induce apoptosis in a p53 2 / 2 lymphoma cell line derived from the mouse model.. Cre-ER

Using the Mx1-Cre;mT/mG reporter mouse model, we found that the calvarial cells exhibited minimal background Mx1-Cre activity prior to Cre activation by IFN α treatment as compared

To compare the normalized data from dysplasia, normal lung tissue from transgenic mouse, tumor cells and non-transgenic of different mice, we used the Significance Analysis

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We generated DLC2-deficient mice to investigate the tumor suppressor role of DLC2 in hepatocarcinogenesis and the function of DLC2 in vivo.. In this study, we found that,

Transgenic mice carrying the human kallikrein gene under the control of the mouse metallothionein metal-responsive promoter expressed the human protein in several or- gans