Abnormalities of hippocampal signal intensity in patients with familial mesial temporal lobe epilepsy

Purpose: To examine temporal pole (TP) signal and quantitative changes in temporal lobe epilepsy (TLE) patients with hippocampal sclerosis (HS) using coronal

Purpose: To examine temporal pole (TP) signal and quantitative changes in temporal lobe epilepsy (TLE) patients with hippocampal sclerosis (HS) using coronal

atrophy (HA) and increased T2 signal in limbic struc- tures are a frequent MRI finding related with mesial temporal sclerosis (MTS) identified in patients with mesial temporal

Furthermore, the MR imaging studies reveal a mass with signal intensity similar to the subcutaneous fat on T1 and T2-weighted images, with chemical-shift artifact around the

Magnetic resonance imaging (MRI) demonstrated the pres- ence of an elongated lesion with regular contour, intermediate signal intensity on T1-weighted and marked hypersignal on

Magnetic resonance imaging (MRI) demonstrated the pres- ence of an elongated lesion with regular contour, intermediate signal intensity on T1-weighted and marked hypersignal on

resonance imaging showed a hypointense signal on a T1-weighted image (Figure 1D) and increased intensity in a T2-weighted short- tau inversion-recovery sequence, with bone

A Mayo Clinic series of 26 patients with LGI1 encephalitis and FDBS showed that basal ganglia T1 and T2 signal abnormalities were detected in 42% of these patients, including